Glutathione is such a critical soldier in our line of defense that our bodies make it themselves. We even have mechanisms that recycle and reuse it. But what happens when our toxic Inhibiting acidsensing ion channel exerts neuroprotective effects in experimental epilepsy via suppressing ferroptosis Shi 2024 CNS Neuroscience & Therapeutics Wiley Online Library Ferroptosis as a therapeutic target in glioblastoma: Mechanisms and emerging strategies: Molecular Therapy Nucleic Acids Pharmacological elevation of glutathione inhibits status epilepticus induced neuroinflammation and oxidative injury ScienceDirect